effect of N acetyl cysteine in intracerebroventricular colchicine induced cognitive deficits, beta amyloid pathology, and Glial cells: research article

among the many factors responsible for the cognitive decline in Alzheimer's disease, beta amyloid protein and plaque formation is crucial. the antioxidant NAC has reversed the cognitive deficits and inhibits the microglial activation. it can be postulated that NAC might have reversed the effect of intraneuronal beta amyloid protein by acting on some downstream compensatory mechanism which needs to be explored.