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Mitochondria–lysosome crosstalk in GBA1-associated Parkinson’s disease
M. Sahyadri
, Abhishek P.R. Nadiga
, Seema Mehdi
, K. Mruthunjaya
,
Pawan G. Nayak
, Vipan K. Parihar
, S. N. Manjula
*
*
Corresponding author for this work
Department of Pharmacology, Manipal College of Pharmaceutical Sciences, Manipal
Manipal College of Pharmaceutical Sciences, Manipal
Research output
:
Contribution to journal
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Review article
›
peer-review
5
Citations (Scopus)
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Biochemistry, Genetics and Molecular Biology
Enzymatic Hydrolysis
100%
Lysosome
100%
Homeostasis
100%
Enzyme Activity
100%
Mitochondrion
100%
Gene Mutation
100%
Cell Fate
100%
Glucosylceramidase
100%
Guanosine Triphosphate
100%
INIS
nervous system diseases
100%
mutations
37%
neurons
37%
mitochondria
37%
organelles
37%
risks
25%
reviews
25%
connections
25%
patients
12%
control
12%
diseases
12%
proteins
12%
genes
12%
interactions
12%
pathogenesis
12%
investigations
12%
humans
12%
hydrolysis
12%
enzyme activity
12%
genetics
12%
homeostasis
12%
mutants
12%
lysosomes
12%
Neuroscience
Parkinson's Disease
100%
Neurodegenerative Disorder
12%
Lysosomes
12%
Mitochondrion
12%
Mitochondrial Function
12%
Cell Fate
12%
Gene Mutation
12%
Lysosome Enzyme
12%
Glucosylceramidase
12%
Guanosine Triphosphate
12%